By Jeffrey B. Halter, Joseph G. Ouslander, Stephanie Studenski, Kevin P. High, Sanjay Asthana, Mark A. Supiano, Christine S. Ritchie

Medication is an ever-changing technology. As new learn and scientific adventure increase our wisdom, alterations in remedy and drug treatment are required. The authors and the writer of this paintings have checked with resources believed to be trustworthy of their efforts to supply info that's whole and customarily in accord with the factors approved on the time of booklet. although, in view of the possibility Read more...


Combining gerontology ideas with medical geriatrics, this identify provides evidence-based clinical info that makes discovering the solutions on your questions quicker and easier. Read more...

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Although most of the published studies, and those in progress, focus on nuclear genes, there are tantalizing suggestions from studies of large families that a surprisingly high proportion of inherited effects on lifespan may come through the maternal line alone, suggesting that variations in mitochondrial gene sequences (which are inherited almost entirely from mothers to both sons and daughters) may also influence life expectancy in humans. A major problem with all these approaches, from the perspective of biological gerontology, is the lack of a defensible phenotype: a measure of aging better than lifespan.

The bottom line shows projected longevity if it were possible to retard human aging to the same degree that is obtained in the typical study of caloric restriction in rats or mice. Figure reprinted with permission from Miller RA. Extending life: scientific prospects and political obstacles. Milbank Quart 2002;80:155–174. But the pathway connecting discovery in this area of basic science to intervention is strewn with obstacles, some scientific, but others political, economic, and legal. Even if research in mice or rats were to demonstrate a dramatic effect of a specific agent on the aging process, producing (for example) a 40% increase in mean and maximal lifespan, showing that the agent was able to safely extend human lifespan would require vast resources and treatment of young or middle-aged adult subjects for many decades.

LD50 is the amount of peroxide that kills 50% of the cells. (D) Resistance of calorie-restricted (Cal-R) mice to liver injury induced by injection of acetaminophen (APAP); serum LDH levels indicate the level of damage to hepatocytes at varying intervals after a single injection (unpublished data of Chang and Miller). (A) Reproduced with permission from Gems D, Sutton AJ, Sundermeyer ML, et al. Two pleiotropic classes of daf-2 mutation affect larval arrest, adult behavior, reproduction and longevity in Caenorhabditis elegans.

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