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By R. van Furth (auth.), Ralph van Furth (eds.)
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E. Cross (1971) The origin of pulmonary alveolar macrophages. Archives of Internal Medicine 127, 1064. A. H. Waksman (1964) The pathogenesis of adjuvant disease in the rat. II. A radioautographic study of early lesions with use of 3H-thymidine. Yale Journal of Biology and Medicine 37, 195. Bursuker, I. & R. Goldman (1979) Derivation of resident and inflammatory peritoneal macrophages from precursor cells differing in 5'-nucleotidase activity. Journal of the Reticuloendothelial Society 26, 205. Carr, I.
Sensitized) lymphocytes, such as lymphokine and interferon, or by serum proteins, for example immunoglobulins and complement components. Stimulatory substances can also lead to activation of mononuclear phagocytes in vitro (0gmundsd6ttir & Weir 1980). All this means that the cells in an inflammatory exudate can be quite heterogeneous, and should therefore be characterized cautiously. Table 1 offers a guideline for their identification. To indicate the degree of heterogeneity of the population of cells in the exudate, however, it is essential to report not only qualitative but also quantitative data for each characteristic.
Carr, I. (1975) The reticular cell, the reticulum cell, and the macrophage. In: van Furth, R. ) Mononuclear phagocytes in immunity, infection, and pathology, p. 119. Blackwell Scientific Publications, oxford-LondonEdinburgh-Melbourne. V. G. Gorynova (1965) In vitro lysozyme formation by culture of histiocyte macrophages. Federation Proceedings; Federation of American Societies for Experimental Biology 25, T707. J. (1978) Multinucleated giant cells. Journal of Pathology 126, 125. A. (1970) Endocytosis and intracellular digestion.